Forms of periodontitis: localized juvenile periodontitis

16-09-2009
Forms of periodontitis: localized juvenile periodontitis
Localized juvenile periodontitis (LJP) – an inflammatory disease that occurs in adolescents aged 10-13 years. For this form of periodontitis is characterized by intensive periodontal lesion with rapidly progressive destruction of bone tissue. High intensity of destruction at the beginning of the disease may continue to slow down or disappear.

The clinical picture of localized Youth periodontitis periodontal lesions characterized mainly in the central incisors or first molars, usually located symmetrically.

Generalized form of this type of periodontitis is rare. It is supposed to move in a localized form of generalized, especially if left untreated, resulting in a short period (1-2 years) progression of the disease can lead to almost complete loss of the restraint apparatus of affected teeth. Gingival tissue, usually shows no or minor signs of inflammation. The presence of a small number of over-and poddesnevogo plaque and tartar are not due to a high degree of periodontal destruction.

Therefore, adolescent patients with clinically healthy periodontium during the inspection should be required to conduct sensing gingival sulcus in the area of molars and incisors. With timely diagnosis and treatment of juvenile periodontitis further course of the disease may be beneficial.

Most patients with this form of periodontitis plaque slight, with a predominance of Actinobacillus actinomycetem comitans and different types of Capnocytophagas. On the origin and development of juvenile periodontitis affect most bacteria Actinobacillus actinomycetem comitans, which penetrates deeply into the soft tissues. Bacteria of this type do not occur in all patients with juvenile periodontitis, ie, no specific infection. The majority of patients with juvenile periodontitis in the survey show a dysfunction of neutrophil granulocytes and monocytes. These blood cells have a reduced ability to chemotaxis and phagocytosis. Moreover, granulocytes philous granulocytes not phagocytose bacteria. It is believed that these defects are inherited by leukocytes autosomal recessive or dominant pattern.

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